Chromosomal Microarray (CMA / aCGH)

Definition

A high-resolution test that detects copy number variations (CNV) across the genome, that is, deletions and duplications too small to be seen under a microscope. It is also called "molecular karyotyping".

Comparison with karyotype, International Consensus (ISCA) data:

Microarray (CMA)G-banded Karyotype
Diagnostic yield15–20%~3% (excluding Down syndrome and recognizable syndromes)
StrengthHigh sensitivity for submicroscopic deletions/duplicationsSees the whole chromosome structure
What it cannot seeTruly balanced rearrangements and low-level mosaicismSmall (submicroscopic) changes

Conclusion of the consensus:

Current evidence strongly supports the use of CMA in place of G-banded karyotype as the first-tier cytogenetic diagnostic test in patients with unexplained developmental delay/intellectual disability, autism spectrum disorder or multiple congenital anomalies.

When karyotype is preferred:

  • If an obvious chromosomal syndrome is suspected (e.g. Down syndrome)
  • If there is a family history of chromosome rearrangement
  • If there is a history of recurrent pregnancy loss

This last point is important: microarray cannot see balanced translocations, yet the main genetic cause of recurrent pregnancy loss is precisely balanced translocation carrier status (see the Chromosome Abnormalities entry). This is why a karyotype is requested in recurrent pregnancy loss.

Relationship with FISH: FISH is a targeted test; it looks at a single predetermined region (e.g. 22q11 DiGeorge, 15q11 Prader-Willi) and gives a rapid result. Microarray, on the other hand, scans genome-wide and requires no target. If the clinical suspicion points to a specific syndrome, FISH is appropriate; if it is unclear, microarray is appropriate.

Risks and Limitations

The balanced rearrangements and low-level mosaicism that CMA cannot see are relatively rare causes of an abnormal phenotype in this patient group (<1%), so the limitation is real but leaves only a small gap in practice.

CMA can generate a large number of VUS; the clinical significance of some copy number variations is uncertain.

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